Contact: Dr. Peter Herter
peter.herter@mpi-dortmund.mpg.de
49-231-133-2500
Max-Planck-Gesellschaft
Caption: The Rab1 protein in its "normal" form (left) and following modification with adenosine monophosphate (AMP; right) using the bacterial enzyme DrrA. The green area shows the contact surface for proteins that convert Rab1 from its active to inactive form. Their attachment is prevented by the AMP residue.
Credit: Max Planck Institute of Molecular Physiology
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