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Contact: Jules Asher
NIMHpress@nih.gov
301-443-4536
NIH/National Institute of Mental Health
Caption: Forebrain of a mouse genetically engineered to express the mutated gene that causes Timothy syndrome (TS) shows fewer neurons contributing to a brain structure responsible for long-distance communications between the left and right hemispheres, called the corpus callosum, compared to the same structure in a control animal (Ctrl). Human iPSCs from TS patients showed a similar reduction.
Credit: Ricardo Dolmetsch, Ph.D., Stanford University
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