![[ Back to EurekAlert! ]](http://www.eurekalert.org/images/back2e.gif){kind=small}
|
Contact: Professor Paul Jarvis
rpj3@leicester.ac.uk
44-011-622-31296
University of Leicester
Caption: This shows chloroplasts from three different genotypes of Arabidopsis thaliana: a healthy wild-type chloroplast with an extensive thylakoid membrane network (center); a chloroplast from a ppi1 mutant plant which lacks the atToc33 protein (bottom); and a ppi1 plant with a second mutation in the SP1 gene (top). Chloroplasts from the sp1 ppi1 double mutant are much more developed than ppi1 single-mutant chloroplasts as the effects of atToc33's absence have been substantially suppressed by the absence of SP1.
Credit: Graphics: Mats and Paula Töpel
Usage Restrictions: Use only with credit.
Related news release: Could chloroplast breakthrough unlock key to controlling fruit ripening in crops?
|