A new study on Alzheimer's disease by Scripps Research scientists has revealed a previously unknown biochemical cascade in the brain that leads to the destruction of synapses, the connections between nerve cells that are responsible for memory and cognition.
According to an article in ARRS' American Journal of Roentgenology (AJR), a prototype 16-channel head Adaptive Image Receive (AIR) radiofrequency coil from GE Healthcare outperformed a conventional 8-channel head coil for in vivo whole-brain imaging, though it did not perform as well as a conventional 32-channel head coil.
Jessica Huber, a professor of Speech, Language, and Hearing Sciences and associate dean for research in Purdue's College of Health and Human Sciences, leads Purdue's Motor Speech Lab. Huber and her team are now doing virtual studies to evaluate speech disorders related to Parkinson's using artificial intelligence technology platforms.
Rice University biochemists have proposed that degenerative diseases as varied as Alzheimer's, Parkinson's and muscle atrophy occur in two distinct phases marked by protein signaling changes that could result in patients responding differently to the same treatment.
A simple eye exam combined with powerful artificial intelligence (AI) machine learning technology could provide early detection of Parkinson's disease, according to research being presented at the annual meeting of the Radiological Society of North America (RSNA).
Though neurodegenerative diseases are becoming more common in today's aging societies, the exact way in which accumulated abnormal proteins become toxic to neurons is unknown. In a recent study conducted at Daegu Gyeongbuk Institute of Science and Technology, Korea, scientists have discovered a new mechanism of action by which these abnormal proteins actually unlock the normally latent toxicity of native proteins. Their results represent a completely new avenue toward the development of effective therapies.
Research from UCLA scientists and colleagues from other institutions finds that people with Parkinson's disease who lack meaningful social interactions may be at an increased risk for severe symptoms related to the disease.
Carnegie Mellon University researchers have developed a new technique for isolating a type of brain cell associated with Parkinson's disease symptoms, enabling them to study that cell type in detail.
Stroke is a leading cause of death and severe long-term disability with limited treatment available. A research team led by Prof. Gong Chen at Jinan University, Guangzhou, China recently reported the first non-human primate study demonstrating successful in vivo neural regeneration from brain internal glial cells for stroke repair. This work was published on Frontiers in Cell and Developmental Biology on November 5th, 2020.
In a new study led by the Immune Systems Biology research group of the LIH Department of Infection and Immunity, researchers adopted a holistic machine-learning approach to elucidate how the interactions between neuronal mitochondria can serve as a powerful tool to distinguish nerve cells from Parkinson's patients from those belonging to healthy subjects, thereby providing new insights in the pathogenesis, diagnosis and treatment of this neurodegenerative disorder. The results were published in 'Nature Partner Journals Systems Biology and Application'.