Artwork depicts KIBRA-dependent recovery of functional plasticity at synapses despite tau-induced toxicity in the brain. (IMAGE)

Buck Institute for Research on Aging

Artwork depicts KIBRA-dependent recovery of functional plasticity at synapses despite tau-induced toxicity in the brain.

Caption

Kauwe, Pareja-Navarro et al. identify a KIBRA-dependent mechanism to repair the plasticity at synapses that is dysregulated in neurons underlying the loss of memory in tauopathy. The art depicts the recovery of the functional plasticity at synapses on neurons despite tau-induced toxicity in the brain.

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Larissa Brown

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