Figure. KEAP1-deficient lung cancer cells use PC-dependent metabolism as a metabolic escape route during KRAS G12C inhibition. Blocking PC restores sensitivity to treatment. (IMAGE)

Kanazawa University

Figure. KEAP1-deficient lung cancer cells use PC-dependent metabolism as a metabolic escape route during KRAS G12C inhibition. Blocking PC restores sensitivity to treatment.

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Figure. KEAP1-deficient lung cancer cells use PC-dependent metabolism as a metabolic escape route during KRAS G12C inhibition. Blocking PC restores sensitivity to treatment.

 

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