Figure 4. (IMAGE)

Tokyo Metropolitan Institute of Medical Science

Figure 4.

Caption

(A) Interaction sites between OPTN and RAB1A/RAB1B.

(B) Proposed model showing how the OPTN–RAB1 interaction promotes recruitment of ATG9A vesicles and the initiation of autophagic membrane formation around damaged mitochondria.

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©Tokyo Metropolitan Institute of Medical Science, or ©TMIMS

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Source of the figure: Published on the official website of the Tokyo Metropolitan Institute of Medical Science.

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