News Release

Study reveals unique genetic architecture of hypertrophic cardiomyopathy in Chinese population

Peer-Reviewed Publication

Higher Education Press

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In this cross-sectional study including 1 825 patients with HCM, Chinese patients have a higher proportion of rare variants but have a similar proportion of likely pathogenic or pathogenic variants compared with Europeans. In addition, c.3624del in MYBPC3 and c.300C>G in TNNT2 are predominantly present in patients of East Asian ancestry. In this study, ethnic disparities of HCM genetic architecture were observed, and a lack of prior research in Chinese populations limits variant interpretation in Chinese ancestry with HCM.

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Credit: Jie Wang, Dominic Russ, Yongsan Yang, Lutong Pu, Mengdi Yu, Jinquan Zhang, Jiajun Guo, Yuanwei Xu, Ke Wan, Heng Xu, Yuchi Han, Georgios V Gkoutos, Yucheng Chen

Hypertrophic cardiomyopathy (HCM), a common genetic heart disorder, is often caused by mutations in sarcomere-related genes. While extensively studied in European populations, its genetic basis in Chinese individuals remains poorly understood.

In a groundbreaking cross-sectional study, researchers from West China Hospital and the University of Birmingham analyzed whole-exome sequencing data from 593 Chinese and 1,232 UK HCM patients, along with controls. They found that Chinese patients carry a significantly higher burden of rare variants (52.8% vs. 13.6% in the UK), yet the proportion of pathogenic or likely pathogenic (P/LP) variants was similar between the two groups.

Notably, two mutations—MYBPC3 c.3624del and TNNT2 c.300C>G—were identified as specific to the Chinese cohort, accounting for 2.9% and 1.5% of cases, respectively. The study also uncovered stronger associations with thin filament and myosin light chain genes in Chinese patients, while MYBPC3 non-truncating variants were more prominent in the UK cohort.

Using the tool genebe, researchers reduced the rate of variants of uncertain significance (VUS) to 46.8%, outperforming other classification tools and improving diagnostic clarity.

These findings underscore the importance of ethnicity-specific genetic databases and refined interpretation frameworks to avoid misclassification and enhance clinical management of HCM across diverse populations.


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