What happens if cancer cells lose both their mitochondrial energy supply and their glycolytic backup system? This question is central to a new study published in Research by researchers from Northwestern Polytechnical University and collaborating institutions. The work addresses a key limitation of cuproptosis-based cancer therapy: copper-dependent mitochondrial stress can kill cancer cells, but many tumors are metabolically flexible and rely heavily on aerobic glycolysis to survive mitochondrial damage. The study proposes a way to block both routes at once.

In a new study published in Research, researchers from Northwestern Polytechnical University and collaborating institutions report a molecular strategy that addresses this problem from within the perovskite layer rather than simply blocking UV light from outside.

A new review published in Research by researchers from Zhejiang Provincial People's Hospital and collaborating institutions focuses on peripheral nerve–cancer interactions and proposes a three-dimensional framework for understanding this emerging field.