(IMAGE)
Caption
Schematic illustration of the luteolin nanomedicine targeting CD44 receptors on macrophage- and smooth muscle cell–derived foam cells within atherosclerotic plaques. Once internalized, the nanomedicine undergoes stimulus-responsive traceless release under oxidative stress and acidic pH, promoting lipid efflux and reducing foam cell burden. This process contributes to plaque reduction and stabilization.
Credit
Dr. He Huang from Zhejiang University, China
Usage Restrictions
Credit must be given to the creator.
License
CC BY