Patients with Long COVID forced to become their own doctors
Peer-Reviewed Publication
Updates every hour. Last Updated: 1-Sep-2025 22:11 ET (2-Sep-2025 02:11 GMT/UTC)
Vaccines trigger a notably rapid response in the stromal cells of draining lymph nodes within the first hours after administration. Researchers at the University of Turku and the InFLAMES Flagship in Finland also discovered that the stromal alterations prime the lymph node landscape for the subsequent steps of vaccine-induced immune responses.
A Covid infection, particularly in women, may lead to blood vessels aging around five years, according to research published in the European Heart Journal [1]. Blood vessels gradually become stiffer with age, but the new study suggests that Covid could accelerate this process. Researchers say this is important since people with stiffer blood vessels face a higher risk of cardiovascular disease, including stroke and heart attack.
The antibody also has potential to treat a broad array of other conditions, including autoimmune disorders, cancer and diabetes, research indicates.
People who have had COVID-19 are at increased risk of developing certain inflammatory diseases of the airways, such as asthma, hay fever and chronic sinusitis. However, vaccination against the SARS-CoV-2 virus appears to reduce the risk, according to a comprehensive epidemiological study led by researchers at Karolinska Institutet.
As the nation gears up for the rollout of an updated COVID-19 vaccine, a new study shows the economic benefits of continued broad vaccination in adults. In fact, the country would ultimately save more money that it would spend on vaccinating every person over age 65 with a single dose of an updated mRNA vaccine against coronavirus, the study concludes.
Public health officials had an unprecedented tool for near-instant, widespread communication during the COVID-19 pandemic and mpox epidemic: social media.
Now, one of the first studies of its kind, led by a health policy expert with the Texas A&M University School of Public Health, has found that X (Twitter at the time of the events) brought advantages — as well as disadvantages — in getting the word out.
Key Findings:
AI-powered Aging Clock: The new proteomic clock predicts biological age with high accuracy (R²=0.84, MAE=2.68 years) and captures accelerated aging signatures in severe lung disease cases—patients with severe COVID-19 (and likely fibrosis) exhibited biological ages nearly three years older than healthy controls.
Distinct Molecular Signatures: Analysis with the ipf-P3GPT generative model revealed both shared and unique gene expression patterns between aging lungs and fibrotic disease, highlighting that IPF is not just accelerated aging but entails unique pathological processes.
Pathway-Level Insights: The study identified four key pathways (TGF-ß signaling, oxidative stress, inflammation, ECM remodeling) as central to both IPF and aging, but involved differently at the gene level.
Groundbreaking research led by a Swansea University academic has revealed a synthetic glycosystem — a sugar-coated polymer nanoparticle — that can block Covid-19 from infecting human cells, reducing infection rates by nearly 99%.
Researchers from the Translational Genomics Research Institute (TGen), part of City of Hope, and the Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center have identified a potential biomarker for long COVID.
If the findings of their study are confirmed by other research centers, the biomarker could be the first specific and quantifiable indicator for confirming long COVID. Currently, clinicians confer a diagnosis of long COVID based upon a collection of symptoms that patients develop after SARS-CoV-2 infection.