Wearable sweat sensors represent a revolutionary shift in ICU monitoring, transitioning from invasive, intermittent blood tests to non-invasive, continuous real-time biochemical tracking. By analyzing electrolytes, metabolites, and hormones, these sensors provide high-resolution physiological data crucial for early sepsis warning and precise metabolic management. Despite challenges like biofouling and complex blood-sweat correlations, integration with digital health systems and AI-driven predictive models promises a future of highly personalized, proactive, and automated critical care.

A team led by Professor Jianping Jia from Xuanwu Hospital of Capital Medical University published significant findings in the Chinese Medical Journal. They developed a novel blood test using ultrasonic protein misfolding cyclic amplification technology. By detecting plasma amyloid beta aggregation seeding activity, this method accurately diagnoses Alzheimer’s disease and mild cognitive impairment. Achieving over 90 percent diagnostic accuracy, it provides an important minimally invasive tool for early and differential diagnosis of the disease.

An international collaborative team led by researchers from the Institute of Physics, Chinese Academy of Sciences, University of Chinese Academy of Sciences and Columbia University has delivered a comprehensive microscopic portrait of magnetic ordering and spin dynamics in Li(Zn,Mn)As, a landmark bulk diluted magnetic semiconductor (DMS) promising to overcome longstanding limitations of classic (Ga,Mn)As thin-film systems. Published in Materials Futures, the study leverages multi-geometry muon spin rotation/relaxation/resonance (µSR) to resolve three core unresolved puzzles surrounding the material’s ferromagnetism, offering quantitative benchmarks for bound magnetic polaron (BMP) percolation theories widely applied to DMS research.

Research teams from The University of Hong Kong, led by Professor Chunyi Zhi, have provided a focused review of aqueous Zn-based flow batteries for long-duration energy storage. The review identifies Zn anode reversibility loss and active-species crossover/shuttle as two coupled bottlenecks, and summarizes design strategies that integrate electrolyte regulation, interfacial engineering, electrode architecture, species chemistry, and membrane design.

Recently, a research team led by Prof. XU An at the Hefei Institutes of Physical Science of the Chinese Academy of Sciences, has demonstrated that the magnetotactic bacterium (MTB) Magnetospirillum magneticum AMB-1 (AMB-1) extended healthy organismal lifespan in the Caenorhabditis elegans model, and uncovered the core mechanism underlying this longevity effect — ferroptosis inhibition.

A unified scaling analysis gives quantum geometry and disorder their own measurable weight patterns, allowing researchers to separate competing sources of the second-order nonlinear Hall effect in existing experiments.

Type 2 diabetes (T2D) develops through the long-term interplay of genetic predisposition, lifestyle, and environmental exposures. Residential greenness is increasingly studied as a measurable environmental exposure that may be related to diabetes risk. However, prospective evidence from Chinese populations remains limited, and whether genetic susceptibility modifies the greenness–T2D association, particularly in terms of absolute risk reduction, has remained unclear.

Metabolic dysfunction-associated steatotic liver disease (MASLD), which affects about 30% of the population worldwide, is driven in part by excessive hepatic de novo lipogenesis (the production of fatty acids within the liver). In mice, the minor intron splicing factors ZRSR1 and ZRSR2 can influence hepatic lipid metabolism by controlling the processing of Insig transcripts. Whether these proteins also regulate metabolism independently of their canonical splicing function has remained unclear.

Chronic kidney disease (CKD) affects millions worldwide, with renal fibrosis representing a final common pathway of chronic renal injury. Defective fatty acid oxidation (FAO) in renal tubular epithelial cells has been shown to promote kidney fibrosis, but whether FAO impairment in kidney vascular endothelial cells contributes to renal pathology has remained unclear.