For the first time since 2018, a clinical guideline from the American College of Cardiology and the American Heart Association for screening and managing blood cholesterol levels has been updated and jointly published in the Journal of the American College of Cardiology and Circulation. The new guideline will be discussed March 28 at the American College of Cardiology’s 75th Annual Scientific Session in New Orleans.

 

The guideline’s release also ran a week before a paper titled “The ABCs of Cardiovascular Disease Prevention: Communicating What We Know in 2026” published in the American Journal of Preventive Cardiology.

The American Heart Association has awarded 32 Nation of Lifesaver™ financial grants to American Heart Association Heart Clubs at high schools and colleges in 20 states, including the District of Columbia.

A new study shows the two cardiovascular conditions may be less distinct than previously thought, intertwining at genetic and mechanistic levels.

A new study from Penn State College of Medicine found that teens who went to sleep later and woke up later were more likely to consume more calories, snack more and be less physically active, suggesting that sleep is a risk factor for cardiometabolic health.

A new consortium, bringing together more than 60 partners from 25 countries, has kicked off a five-year initiative to help improve the heart health of people living with and beyond cancer with a two-day meeting at St Thomas' Hospital and the London Institute for Healthcare Engineering.

LMU volcanologists decipher the behavior of magma beneath an active volcano and reveal how it reacts to drilling.

Down syndrome (DS), caused by trisomy 21 (T21), affects approximately 1 in 700 live births and is characterized by a wide spectrum of features, including cognitive impairment, craniofacial abnormalities, congenital heart defects, and early-onset Alzheimer disease (EOAD). Among the ~364 protein-coding genes triplicated on HSA21, DYRK1A (dual-specificity tyrosine phosphorylation-regulated kinase 1A) has emerged as a key contributor to neurodevelopmental deficits. This review summarizes the neurogenetic consequences of DYRK1A triplication, its interactions with other genes, and therapeutic strategies targeting its overexpression.