Bar-Ilan University Technology helps reveal elusive Alzheimer's protein aggregates missed by current tests
Peer-Reviewed Publication
In honor of Alzheimer's Awareness Month, we’re exploring the science and stories surrounding Alzheimer’s disease.
Updates every hour. Last Updated: 29-Oct-2026 21:16 ET (30-Oct-2026 01:16 GMT/UTC)
A molecular imaging technology developed by Prof. Shai Rahimipour of Bar-Ilan University is helping scientists uncover one of the earliest and most elusive drivers of Alzheimer's disease, opening new possibilities for earlier diagnosis and more precise treatment.
The technology played a central role in a new international study published in Alzheimer's & Dementia: The Journal of the Alzheimer's Association, led by researchers at the Icahn School of Medicine at Mount Sinai in New York together with collaborators from Bar-Ilan University and Canada. The study provides compelling evidence that soluble amyloid-beta oligomers -- tiny toxic protein aggregates believed to trigger Alzheimer's disease years before symptoms appear -- can damage brain cells long before they become detectable by today's diagnostic methods.
A molecule linked to chronic inflammation disrupts the brain’s ability to create new neurons – uncovering a pathway with therapeutic potential for neurodegenerative diseases and neurological conditions.
A research paper by scientists from Chinese Academy of Medical Sciences and Peking Union Medical College applied transcranial photobiomodulation (tPBM) with continuous-wave (CW) and 40-Hz pulsed light to target neurovascular pathology in 5xFAD mice.
The new research paper, published on Jun. 23 in the journal Cyborg and Bionic Systems, has uncovered how two distinct transcranial photobiomodulation (tPBM) protocols–continuous‑wave (CW) and 40‑Hz pulsed light–can tackle both amyloid pathology and vascular impairment through different glial‑mediated mechanisms.
Researchers used machine learning to show how a common tire by-product could disrupt brain function