Agricultural scientists are rethinking their battle plans against plant parasites that cause billions of dollars in annual yield losses for American farmers. 

Chemical nematicides, soil treatments and biological controls have had mixed success against the soybean cyst nematode, and many potential solutions carry environmental or economic limitations.

Host resistance — breeding soybean varieties that can withstand infection — has been the most effective and environmentally friendly management tool, but its usefulness is declining. Asia Kud, an assistant professor of nematology in the department of entomology and plant pathology for the Arkansas Agricultural Experiment Station, the research arm of the University of Arkansas Division of Agriculture, Kud was awarded a $298,913 grant from the U.S. Department of Agriculture’s National Institute of Food and Agriculture to continue researching a strategy to stop these pest nematodes from feeding and reproducing by interfering feeding sites they create with proteins called effectors.

What happens when evolution rewires a brain for life in total darkness? In a study of the blind Mexican cavefish, researchers uncovered an evolutionary surprise: unlike their sighted relatives, cavefish become more active in light rather than darkness. Using whole-brain imaging at cellular resolution, the team revealed that neural circuits have effectively flipped their responses to light and dark. The findings offer a rare glimpse into how evolution reshapes the brain and could shed light on neurological disorders ranging from Parkinson’s disease to autism and ADHD.

This peer-reviewed Perspective synthesizes a decade of research on a surprising candidate for autism science: the laboratory Beagle. Drawing together scattered studies, the review authors argue that dogs carrying engineered changes in the Shank3 gene reproduce core human features of autism spectrum disorder, including social withdrawal, altered sensory responses, and reduced attention to the eyes of a face. The synthesis assembles, for the first time in one place, the parallels between mutant dogs and autistic people, alongside early signals that oxytocin, a psychedelic, and a third compound may soften these traits. The authors frame the dog as a complementary, ethically governed model, not a replacement for existing ones.