Identify birds offline using AI: New app records animal sounds directly on your smartphone
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Updates every hour. Last Updated: 31-Oct-2026 00:16 ET (31-Oct-2026 04:16 GMT/UTC)
Technion researchers have identified a previously unknown mechanism that enables bacteria to develop antibiotic resistance far more rapidly than previously understood. The study, led by Dr. Idan Yelin and Prof. Roy Kishony of the Technion’s Faculty of Biology and published in Nature Microbiology, reveals how bacteria can quickly produce dozens of copies of genes that confer resistance.
Using AmpliFinder, a new computational tool, the researchers analyzed more than 10,000 laboratory-evolved bacterial samples and discovered that non-canonical gene amplifications are both common and highly efficient. Experiments in Escherichia coli and Acinetobacter baumannii showed that this mechanism enables bacteria to adapt to progressively higher concentrations of antibiotics. Disrupting the process could offer a new way to slow the emergence of resistance and improve the effectiveness of antibiotic treatments.
Researchers at IBEC have discovered that epithelial tissues adapt to prolonged stretching by slowly reorganising their keratin cytoskeleton into large multicellular networks. Using advanced live imaging, engineered tissue models and computational simulations, the team found that this process eventually pushes the cell nucleus out of its surrounding keratin cage. The findings reveal a previously unknown mechanism by which tissues respond to sustained mechanical stress, with potential implications for development, organ physiology and diseases linked to defective keratin organisation.
Invasive rats on tropical islands are creating winners and losers among the tiny underwater animals near the bottom of the food chain that inhabit surrounding coral reefs.
Researchers at Lancaster University and the University of Texas say the changes to these marine ecosystems can rewire food chains on the reefs.
Results show that the main mode of seabird nutrients moving up food webs is through fish, which feed both specialised fish-eating predators and more generalised carnivores. As such, when seabird nutrients are added or taken away from ecosystems, there is a rewiring of how energy travels up food webs, and a reconfiguration of larger fish communities.
Researchers from Jiangsu University have identified Pm72, a new powdery mildew resistance gene from the Iraqi wild einkorn wheat accession PI 427741. Genetic analysis showed that the accession carries two dominant resistance genes, including PmNCA6 previously identified on chromosome arm 7AL. Pm72, located on chromosome arm 6AL, provided resistance against all ten powdery mildew isolates tested and remained effective after being transferred into hexaploid wheat through interspecific hybridization. The findings establish Pm72 as a promising resource for breeding wheat varieties with broad and effective resistance to powdery mildew.
Kyoto, Japan -- As close relatives of humans, chimpanzees had sometimes been used in biomedical experiments. They were instrumental in the identification of the hepatitis C virus: the then mysterious pathogen was artificially injected into chimpanzees for study, leading to its eventual identification in 1989. Since then, treatments for humans have advanced and hepatitis C can now be nearly cured with medication. However, the same treatment has not been extended to the chimpanzees.
In 2022, a team of researchers from Kyoto University found that chimpanzees infected with hepatitis C had experienced negative health effects and shorter lifespans. This time, in a collaboration with Kumamoto University, the team was determined to eliminate this virus in chimpanzees residing at Kumamoto Sanctuary, a research facility of Kyoto University’s Wildlife Research Center. The sanctuary is home to several chimpanzees who were once used in medical experiments by multiple organizations in Japan, and is a place where they can spend the rest of their lives being cared for.
"Prioritizing chimpanzee health and welfare is the number one reason for conducting this study," says first author Satoshi Hirata. "We felt that it is our responsibility to do whatever we can to improve their condition."
Metabolic dysfunction-associated steatotic liver disease (MASLD), which affects about 30% of the population worldwide, is driven in part by excessive hepatic de novo lipogenesis (the production of fatty acids within the liver). In mice, the minor intron splicing factors ZRSR1 and ZRSR2 can influence hepatic lipid metabolism by controlling the processing of Insig transcripts. Whether these proteins also regulate metabolism independently of their canonical splicing function has remained unclear.