Herpesvirus protein mimics host enzyme to balance infection and latency
Peer-Reviewed Publication
Updates every hour. Last Updated: 24-Sep-2025 10:11 ET (24-Sep-2025 14:11 GMT/UTC)
Herpesviruses, which cause skin and genital infections, neonatal diseases, and meningitis, can successfully persist over a lifetime and transmit from one host to another. One key strategy that enables them to coexist within host cells is mimicry of cyclin-dependent kinases (CDKs)—proteins that regulate crucial cellular processes. A new study by researchers from Japan reveals novel insights into mechanisms underlying CDK mimicry by viral kinases, driven by phosphorylation of conserved amino acid residues.
Scientists at the Smithsonian Tropical Research Institute (STRI) found that the fringe-lipped bat, known to eavesdrop on frog and toad mating calls to find its prey, learns to distinguish between palatable and unpalatable frogs and toads through experience. The findings, published April 29 in the Proceedings of the Royal Society B, provide the first evidence that eavesdropping predators fine-tune their hunting cues over the course of their development.