Understanding how molecules interact with ions is a cornerstone of chemistry, with applications from pollution detection and cleanup to drug delivery. In a series of new studies led by JILA Fellow and University of Colorado Boulder chemistry professor Mathias Weber, researchers explored how a specific ion receptor called octamethyl calix[4]pyrrole (omC4P) binds to different anions, such as fluoride or nitrate. These findings, published in The Journal of the American Chemical Society, The Journal of Physical Chemistry Letters, and The Journal of Physical Chemistry B, provide fundamental insights about molecular binding that could help advance fields such as environmental science and synthetic chemistry. 

Several prominent phenomena of condensed matter, exemplified by nonlinear transport, flat-band superconductivity, and fractional Chern insulators, are systematically reviewed with a focus on their quantum geometric origins.

Researchers at the Korea Research Institute of Chemical Technology (KRICT), led by Dr. Chi Hoon Park, have successfully generated CAR-M (Chimeric Antigen Receptor Macrophages) by stably inserting synthetic genes into human macrophages derived from peripheral blood using a lentiviral delivery system.

A retrospective cohort study aimed to clarify the association between the number of teeth and the incidence of hip fractures.

Malaria, an infectious disease caused by Plasmodium parasites, is pathologically linked to the dynamic equilibrium of host innate immune responses. Neutrophils, as pivotal effector cells of the innate immune system, constitute the first line of defense against pathogen invasion. Previous work by Professor Qijun Chen's team demonstrated that neutrophils significantly suppress Plasmodium proliferation during early infection through the release of neutrophil extracellular traps (NETs). However, the precise functional specialization and regulatory dynamics of neutrophil subsets during malaria infection have yet to be fully elucidated. Building upon these findings, the team's preliminary investigations revealed that oral gavage administration of dihydroartemisinin (DHA)—a widely deployed antimalarial drug—in murine models induced a marked elevation in the proportion of splenic Ly6G⁺ neutrophils. Nevertheless, the mechanistic basis underlying the antimalarial efficacy of this specific neutrophil subpopulation, particularly its immunomodulatory capacity, remains deep exploration.