What happens to mental health when collective trauma doesn’t end? Lessons from October 7
Peer-Reviewed Publication
Updates every hour. Last Updated: 31-Oct-2026 13:15 ET (31-Oct-2026 17:15 GMT/UTC)
A new systematic review published in Frontiers in Psychiatry brings together 68 studies from the first two years after the October 7 attacks, offering insights that extend far beyond Israel into how prolonged conflict, cumulative exposure and continuing threat can shape post-traumatic stress, anxiety, depression and addiction-related outcomes.
Although there are many cancer treatments available today that effectively eliminate abnormal cells, these therapies may also damage healthy cells, leading to unwanted side effects. Now, researchers publishing in ACS’ Journal of Medicinal Chemistry have developed a new therapeutic that is kept “locked” unless it finds itself within a tumor cell. In mice, the treatment significantly reduced tumor size without accumulating in the liver, which suggests adverse exposures could be reduced and treatment safety improved.
Researcher have identified a sugar structure that corresponds to a component of the C. auris surface. The molecule is a manmade versoion of the same structure that is found in the fungus, and it can be specifically recognised by the immune system, binding in a way that helps to direct the immune response e specifically against the fungus.
The sugar structure serves as the basis for a vaccine candidate and for protective antibodies. In addition, the researchers used one of these antibodies to develop a prototype for a rapid test to detect multiple Candida species in a lateral flow device format – similar to those used to detect the Covid virus..
Calls to poison centers involving liver injuries caused by medications, supplements, alcohol and other substances jumped nearly 400% between 2000 and 2024, according to a new study from UVA Health.
A study of non-functional pancreatic neuroendocrine tumors (NF-PanNETs) identified an AGR2high endocrine subcluster enriched in high-grade tumors and showed that simple AGR2 immunohistochemistry serves as a clinically informative prognostic marker. Mechanistically, the transcription factor FOXM1 directly drives the proliferative transcriptional program of AGR2high cells, while chemical inhibition of FOXM1 showed therapeutic benefit in tumor cells, patient-derived organoids, and xenograft models. Together, the findings provide a new approach for prognostic stratification and identify FOXM1 as a potential therapeutic vulnerability in NF-PanNETs.
Established Alzheimer’s markers are also detected in blood samples from Nigeria and Tanzania. But other proteins in the blood differ from those seen in a comparable group in Canada. This is important if blood tests for Alzheimer’s are to be used globally.